Last week I blogged on the fact that the drug companies were on a campaign to saturate the news media with propaganda that nutrients like C & E had no effect on cardiovascular disease or cancer. I explained that they always use too little or low quality nutrients so the studies come out the way they want them to. Basically they want them to fail. Below I have reprinted a study you all should read. This is why they don't want you to take charge of your own health with natural supplements.
As I have explained before, all animals create ascorbic acid from their livers except, humans, guinea pigs, bats and a few monkeys. It is produced at varying levels depending on the animals stress levels. Vitamin C along with the amino acids (proteins) lysine & proline, are needed by the body to produce collagen, which keeps arteries strong and healthy. Notice what happened in this study when they caused mice to not be able to convert ascorbic acid on their own, like they normally do. The second study used 700 mgs of C which is a little higher than last weeks study, by a third and it had a measurable improvement. Imagine if they had used the levels Linus Pauling said were needed to prevent & reverse cardiovascular disease, and the amount the mouse would naturally produce on it's own, what might have happened.
If you are a thinking individual, a light should go on in your brain as to why conventional medicine and the drug companies, might not want you to know this, or to practice preventative medicine on your own. It's just too powerful.
Stay young as you age...
cw
10639167 (PubMed)
20105560 (MEDLINE)
Title: Aortic wall damage in mice unable to synthesize ascorbic acid.
Authors: Maeda N; Hagihara H; Nakata Y; Hiller S; Wilder J;Reddick R
Journal: Proc Natl Acad Sci U S A
Volume: 97
Issue: 2
Date: 2000 Jan 18
Year: 2000
Pages: 841-6
Review Status: Peer Reviewed
Abstract:
By inactivating the gene for L-gulono-gamma-lactone oxidase, a key enzyme in ascorbic acid synthesis, we have generated mice that, like humans, depend on dietary vitamin C.
Regular chow, containing about 110 mg/kg of vitamin C, is unable to support the growth of the mutant mice, which require L-ascorbic acid supplemented in their drinking water (330 mg/liter). Upon withdrawal of supplementation, plasma and tissue ascorbic acid levels decreased to 10-15% of normal within 2 weeks, and after 5 weeks the mutants became anemic, began to lose weight, and die. Plasma total antioxidative capacities were approximately 37% normal in homozygotes after feeding the unsupplemented diet for 3-5 weeks. As plasma ascorbic acid decreased, small, but significant, increases in total cholesterol and decreases in high density lipoprotein cholesterol were observed. The most striking effects of the marginal dietary vitamin C were alterations in the wall of aorta, evidenced by the disruption of elastic laminae, smooth muscle cell proliferation, and focal endothelial desquamation of the luminal surface. Thus, marginal vitamin C deficiency affects the vascular integrity of mice unable to synthesize ascorbic acid, with potentially profound effects on the pathogenesis of vascular diseases. Breeding the vitamin C-dependent mice with mice carrying defined genetic mutations will provide numerous opportunities for systematic studies of the role of antioxidants in health and disease.
Vitamin C May Reduce CHD Risk
01/03/2005
BOSTON--Vitamin C was inversely associated with risk of coronary heart disease (CHD) in a cohort study that examined the relationship between antioxidants and CHD. The study appears in the
December issue of the American Journal of Clinical Nutrition (80, 6:1508-1520, 2004)
(www.ajcn.org).
The study pooled nine prospective studies containing information on intakes of vitamin E, carotenoids and vitamin C. During a 10-year follow-up, 4,647 CHD events occurred in 293,172 subjects who were free of CHD at baseline.
Subjects with a daily intake of 700 mg or more vitamin C had a lower risk of CHD. Also, dietary intake of antioxidant vitamins was only weakly related to a reduced risk of CHD after adjustment for confounding factors, and supplemental vitamin E intake was not related to lowered CHD risk.
The scientists concluded high vitamin C supplement intake produces a reduced incidence of CHD events, and noted vitamin E and carotenoids apparently do not cause significant reductions in CHD risk.
Christopher Wiechert's Healthblogger is for educational or informational purposes only, and is not intended to diagnose or provide treatment for any condition. If you have any concerns about your own health, you should always consult with a health care professional. If you decide to use this information on your own, it's your constitutional right, but I assume no responsibility. The views expressed on this website are those of the health professionals & scientists I list or my own opinions and are not intended to replace any medical advice you may require. The contents have not been approved by the Pharmaceutical Association, the American Medical Association, or the Food and Drug Administration. This website may present views diametrically opposed to the views of such organizations.
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